What is the purpose of Paclitaxel 260mg Injection?
Paclitaxel 260mg Injection (in conjunction with polyoxyethylated castor oil) is used to treat breast cancer, ovarian cancer (cancer that develops in the female reproductive organs where eggs are produced), and non-small cell lung cancer (NSCLC).
What is Paclitaxel 260mg Injection and how does it work?
Paclitaxel 260mg Injection in conjunction with cisplatin is used to treat non-small cell lung cancer in patients who aren’t candidates for potentially curative surgery or radiation therapy.
How effective is Paclitaxel 260mg Injection?
Many of the participants in these studies had previously undergone chemotherapy. Nonetheless, single-agent therapy with weekly paclitaxel has obtained response rates of up to 86 percent, combination therapy has achieved response rates of up to 87 percent, and radiation has achieved response rates of up to 100 percent.
How do you feel now that you’ve had paclitaxel?
A mild allergic reaction to paclitaxel may occur during or shortly after treatment. Irritation, a rash, or a flushed face are all possible outcomes. A severe allergic reaction is quite uncommon. Symptoms include shortness of breath, swelling of the cheeks and lips, and disorientation.
What are the paclitaxel side effects?
- It is possible to experience nausea,
- mouth sores,
- muscle/joint discomfort,
- numbness/tingling/burning of the hands/feet,
- dizziness, or drowsiness.
- If any of these adverse effects persist or worsen, contact your doctor right once.
- It’s possible that you’ll lose some hair temporarily.
- Normal hair growth should continue after the operation.
Is paclitaxel causing you to lose your hair?
It’s possible that your hair will thin, become brittle, or fall out. This normally occurs two to three weeks after the start of treatment. It is possible to lose all body hair, including pubic, underarm, legs/arms, eyelashes, and nose hairs. Scarves, wigs, caps, and hairpieces may be useful.
Paclitaxel affects which checkpoint in the cell?
The activation of the mitotic checkpoint (also known as the spindle assembly checkpoint), the principal cell cycle regulatory mechanism working during mitosis to avoid chromosomal missegregation, causes paclitaxel-induced mitotic arrest.
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